Friday, November 30, 2012

Grow Your Business With These Simple Marketing With Email Tips ...

E-mail marketing is one of the most effective methods of advertising today. If you?re thinking about getting into email marketing then use all of the tips in this article to help you get started today.

Use the information you already know to create a much better shopping experience. If a customer is filling out an order form linked from an opt-in email, automatically fill in the information you already know about the customer. This saves time and increases the likelihood that your customers will buy something.

In order to cover all of your bases when you market through email, make use of multi-part messages. These emails include both plain and HTML texts, so they will be displayed no matter the option your recipient favors. This is worth the extra step because someone who only wants plain-text will most likely unsubscribe from an email list that is only HTML.

TIP! Use passive and active feedback to make your email marketing better. Active feedback is exactly what you would expect: asking reader opinions in the form of surveys or questionnaires.

If you plan on utilizing email marketing as a promotional strategy, it is crucial that everyone on your subscriber list has authorized you to send them emails. If you do not do this, you will be accused of sending spam, and it may cause you to lose some customers.

Have goals with your audience. Why would your readers want to sign up for your email list? Once you have the answer, use it to your advantage. You should also be sure you have a subscribe link sent out in your email. This will help your existing customers send a link to their friends. This allows your database to grow organically.

Be sure you?ve digested the valuable information detailed in this article. You should either go through these paragraphs again, or bookmark this article for easy access. That way you will have no trouble applying the advice presented here for successfully manifesting your ambitions.

TIP! Keep in mind that the sole purpose of building an email list is to sell your wares. Therefore, every email you send should increase the likelihood that subscribers will buy.

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Source: http://elektrotehnickifakultet.com/grow-your-business-with-these-simple-marketing-with-email-tips.html

davy jones death

Thursday, November 29, 2012

Tiny algae shed light on photosynthesis as a dynamic property

ScienceDaily (Nov. 28, 2012) ? One of the first chemical reactions children learn is the recipe for photosynthesis, combining carbon dioxide, water and solar energy to produce organic compounds. Many of the world's most important photosynthetic eukaryotes such as plants did not develop the ability to combine these ingredients themselves. Rather, they got their light-harnessing organelles -- chloroplasts -- indirectly by stealing them from other organisms. In some instances, this has resulted in algae with multiple, distinct genomes, the evolutionary equivalent of a "turducken*."

Chloroplasts originally evolved from photosynthetic bacteria by primary endosymbiosis, in which a bacterium or other prokaryote is engulfed by a eukaryotic host. The chloroplasts of red and green algae have subsequently come to reside within other, previously non-photosynthetic eukaryotes by secondary endosymbiosis. Such events have contributed to the global diversity of photosynthetic organisms that play a crucial role in regulating and maintaining the global carbon cycle. In most organisms that acquired photosynthesis by this mechanism, the nucleus from the ingested algal cell has disappeared, but in some cases it persists as a residual organelle known as a nucleomorph. Such organisms have four distinct genomes.

To better understand the process of secondary endosymbiosis and why nucleomorphs persist in some organisms, an international team composed of 73 researchers at 27 institutions, including the U.S. Department of Energy Joint Genome Institute (DOE JGI), collaborated to sequence and analyze the genomes and transcriptomes (the expressed genes) of two tiny algae. The team led by John Archibald of Canada's Dalhousie University published their findings on the algae Bigelowellia natans and Guillardia theta ahead online November 29, 2012 in Nature.

Archibald compared these algae to Russian nesting dolls with "sophisticated sub-cellular protein-targeting machinery" and four genomes derived from the two eukaryotes that merged over time. Approximately 50 percent of the genes in both genomes are 'unique' with no obvious counterpart in other organisms," he added. "This indicates just how different they are from characterized species."

DOE JGI Fungal Genomics Program head Igor Grigoriev called B. natans and G. theta "living fossils" because of the remnant nucleomorph. He added that algae are relevant to the Department of Energy Office of Science research portfolio for their potential applications in the fields of bioenergy and environment, noting that the DOE JGI has published over 75 percent of the publicly available algal genomes. "Iterations of endosymbiosis have led to a global diversity of these primary producers. Sequencing these two algae, the first cryptophyte and the first chlorarachniophyte sequenced, helped us to fill in the gaps in the Eukaryotic Tree of Life, and obtain additional references for better understanding of eukaryotic evolution."

Archibald said that "G. theta and B. natans both possess a surprisingly complex suite of enzymes involved in carbon metabolism, and thus represent a useful resource for scientists engaged in both basic and applied research, including photosynthesis, sub-cellular trafficking and biofuels development."

The DOE JGI sequenced the genomes of B. natans (95 million nucleotides or bases: Mb) and G. theta (87 Mb) from single cell isolates provided by Bigelow Laboratory for Ocean Sciences as part of the 2007 Community Sequencing Program portfolio. In addition, the transcriptomes were sequenced separately by the National Center for Genome Resources in New Mexico. Among the team's findings is an answer to the question of why nucleomorphs still exist.

"The reason for the persistence of nucleomorphs in both organisms appears to be surprisingly simple: they are no longer able to transfer their DNA to the host cell nucleus by the process of endosymbiotic gene transfer," said Archibald. Unlike most other secondarily photosynthetic eukaryotes in which the endosymbiont's genetic matter has completely migrated over to the host, in cryptophytes and chlorarachniophytes the nucleus and chloroplast from the engulfed algae remain partitioned off from the host cell. "As a consequence," he added," genetic and biochemical mosaicism is rampant in G. theta and B. natans."

Both researchers highlighted the unexpected finding of alternative splicing in B. natans. Grigoriev noted that the phenomenon is one typical for higher eukaryotes, and Archibald added that the levels "greatly exceed that seen in the model plant Arabidopsis and on par with the human cerebral cortex, unprecedented and truly remarkable for a unicellular organism. This challenges the paradigm that complex alternative splicing is a phenomenon limited to sophisticated multicellular organisms."

"The evolution of chloroplasts, the photosynthetic compartments of plants and algal cells, is complex but has had a profound effect on our planet," said Chris Howe, Professor of Plant and Microbial Biochemistry at Cambridge University in England. "This paper gives us fascinating insights into how host and nucleomorph genomes have been remodeled during evolution. As well as providing a goldmine of information on the general biology of these organisms, the paper shows us that the nucleomorph genomes have probably persisted simply because the mechanism for transfer of genes to the nucleus was closed off, rather than because nucleomorphs had to be retained as separate entities. The paper also shows us that the evolutionary history of the nucleomorph-containing organisms was even more complex than we thought, with evidence for genes from many different sources in their nuclei. Overall, it has provided important insights into the fundamental processes of cell symbiosis and genome reshaping that have produced some of the most important organisms we see today."

*a seasonal dish that consists of a deboned chicken stuffed into a deboned duck, which itself is stuffed into a deboned turkey.

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The above story is reprinted from materials provided by DOE/Joint Genome Institute.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. Bruce A. Curtis, Goro Tanifuji, Fabien Burki, Ansgar Gruber, Manuel Irimia, Shinichiro Maruyama, Maria C. Arias, Steven G. Ball, Gillian H. Gile, Yoshihisa Hirakawa, Julia F. Hopkins, Alan Kuo, Stefan A. Rensing, Jeremy Schmutz, Aikaterini Symeonidi, Marek Elias, Robert J. M. Eveleigh, Emily K. Herman, Mary J. Klute, Takuro Nakayama, Miroslav Oborn?k, Adrian Reyes-Prieto, E. Virginia Armbrust, Stephen J. Aves, Robert G. Beiko, Pedro Coutinho, Joel B. Dacks, Dion G. Durnford, Naomi M. Fast, Beverley R. Green, Cameron J. Grisdale, Franziska Hempel, Bernard Henrissat, Marc P. H?ppner, Ken-Ichiro Ishida, Eunsoo Kim, Lud?k Ko?en?, Peter G. Kroth, Yuan Liu, Shehre-Banoo Malik, Uwe G. Maier, Darcy McRose, Thomas Mock, Jonathan A. D. Neilson, Naoko T. Onodera, Anthony M. Poole, Ellen J. Pritham, Thomas A. Richards, Gabrielle Rocap, Scott W. Roy, Chihiro Sarai, Sarah Schaack, Shu Shirato, Claudio H. Slamovits, David F. Spencer, Shigekatsu Suzuki, Alexandra Z. Worden, Stefan Zauner, Kerrie Barry, Callum Bell, Arvind K. Bharti, John A. Crow, Jane Grimwood, Robin Kramer, Erika Lindquist, Susan Lucas, Asaf Salamov, Geoffrey I. McFadden, Christopher E. Lane, Patrick J. Keeling, Michael W. Gray, Igor V. Grigoriev, John M. Archibald. Algal genomes reveal evolutionary mosaicism and the fate of nucleomorphs. Nature, 2012; DOI: 10.1038/nature11681

Note: If no author is given, the source is cited instead.

Disclaimer: Views expressed in this article do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/top_news/top_environment/~3/V0cnVUjbw0w/121128132253.htm

nicollette sheridan

Wednesday, November 28, 2012

Germany displaces China as US Treasury's currency villain

Germany's permanent surplus is in stark contrast to the shift under way in Asia. China has "partially succeeded in shifting away from a reliance on exports for growth", and has slashed its surplus to 2.6pc from 10.1pc in 2007.

While the yuan remains "significantly overvalued", China?s has stopped building reserves to hold down it currency and has seen a 40pc appreciation against the dollar since 2005 in real terms. Double-digit wage growth is closing thecurrency gap by oither means.

A chart published in the report shows that Germany has overtaken China to become the biggest single source of global trade imbalance, alone accounting for a large chunk of the US deficit.

Switzerland is top sinner with a surplus of 13pc GDP, though the report says the country faces unique circumstances as a safe-haven battling deflation.

The Swiss National Bank has bought $230bn in foreign bonds since mid 2011 to hold the franc, more than China, Russia, Saudia Arabia, Brazil and India combined.

The US Treasury?s shift in focus away from China - and towards Germany?s disguised mercantilism - reflects mounting irritation in Washington over North Europe?s "free-rider" strategy, which relies on exploiting global demand rather than generating it at home.

The US Treasury said China still needs to do more to wean itself off investment - almost 50pc of GDP - and boost consumption instead. It called for a change in the tax structure, reform of the big state enterprises, and an end to financial controls that force up the savings rate. There is concern that China?s surplus will rise again over coming years unless Beijing pushes through radical reforms.

The tone of the report is conciliatory, a far cry from the hot rhetoric of the US election campaign. Republican candidate Mitt Romney had vowed to label China a currency manipulator from "day one", a move that would have entailed trade sanctions and an ugly turn in superpower relations.

A separate report from the International Monetary Fund said China?s excess credit growth and investment have moved into "dangerous territory" and has begun to impose major costs on China itself.

The country spending 10pc of GDP more on investment than the Asian tigers at the peak of the investment bubble before onset of the East Asian meltdown in the late 1990s.

The Fund said the excesses are unlikely to lead to the sort of sudden-stop crisis seen in Thailand, Indonesia and Korea during that episode, since those countries relied on dollar funding whereas China?s credit comes from internal savings, but there is disguised damage nevertheless. Rampant over-investment acts through complex channels as a transfer of income from families and small businesses to big state firms, distorting the whole economic system over time.

The IMF said there is little doubt that investment in plant and infrastructure has driven China?s great boom over the last thirty years but the law of diminishing returns is setting in.

"The marginal contribution of an extra unit of investment to growth has been falling, necessitating ever larger increases to generate an equal amount of growth. Now with investment to GDP already close to 50 percent, the current growth model may have run its course," it said.

Source: http://telegraph.feedsportal.com/c/32726/f/568423/s/2610c7c9/l/0L0Stelegraph0O0Cfinance0Ccurrency0C970A98820CGermany0Edisplaces0EChina0Eas0EUS0ETreasurys0Ecurrency0Evillain0Bhtml/story01.htm

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Report: Kabul Bank sent millions of dollars abroad

KABUL, Afghanistan (AP) ? Hundreds of millions of dollars from Kabul Bank were spirited out of Afghanistan ? some smuggled in airline food trays ? to bank accounts in more than two dozen countries, according to an independent review released on Wednesday about massive fraud that led to the collapse of the nation's largest financial institution.

The report, which was financed by international donors, offers new details about how the men at Kabul Bank and their friends and relatives got rich off $861 million in fraudulent loans in what the International Monetary Fund has called a Ponzi scheme that used customer deposits and operated under nascent banking oversight in the war-torn country.

The report describes Kabul Bank as a sophisticated operation with one set of books for the eyes of regulators and another in the back room that logged how those running the bank and others were fattening their wallets.

Loans were made, but rarely repaid. Borrowers took out loans to pay back loans. Company documents and financial statements were fabricated. The bank's credit department used more than 100 corporate stamps for fake companies to make documents look authentic. The bank operated some of its more than 100 branches without a permit from the government.

The 87-page report, which was conducted to satisfy one of several benchmarks the IMF asked the Afghan government to meet in cleaning up the scandal, points to poor oversight by Afghan banking regulators, political interference in the criminal investigation and activities by a special judicial tribunal hearing the case that it said were "well outside the legal norms of criminal procedure."

The bank's failure and subsequent bailout represents more than 5 percent of Afghanistan's gross domestic product, making it "one of the largest banking failures in the world," according to the report by the Independent Joint Anti-Corruption Monitoring and Evaluation Committee. The report said "hundreds of millions" were sent out of the impoverished nation where Afghan, U.S. and NATO forces are fighting an 11-year-old war with the Taliban and other militants.

"Every citizen in Afghanistan will bear the cost of the hundreds of millions of dollars required to secure deposits and the tens of millions of dollars required to deal with the aftermath," the report said. "This is real money from the annual budget of the government that could be much better spent on other priorities, such as education, health care, infrastructure or security."

The Kabul Bank scandal is a saga about money-grabbing, weak banking oversight, lax prosecution, nepotism, political contributions and fraud. The cast of characters includes a poker-playing bank chairman, an Afghan central bank chairman who feared his life was endangered and fled to the U.S., the wealthy brothers of the Afghan president and vice president, and bank shareholders ? some who bought posh properties in Dubai and spent lavishly on themselves and their circle of friends and relatives.

President Hamid Karzai announced in April 2011 that Kabul Bank would be put into receivership. Earlier this month, a trial began for more than 20 people indicted in the debacle, which has become a symbol of the country's deep-rooted corruption and cronyism. The case is being closely followed by Afghans and international donors because it is a barometer of government officials' pledge to root out patronage, graft and show accountability to international donors.

The report said Afghan authorities learned in late 2009 that "Kabul Bank was moving money through food trays" on flights operated by Pamir Airways, a multimillion-dollar Afghan airline that was established with loans from the bank and has since gone out of business.

An official knowledgeable about the report said as much as $900 million ? a majority derived from loan schemes ? was moved out of the country through electronic transfers between March 2007 and April 2011. That money, the official said, ended up in bank accounts in 28 countries, including the United Arab Emirates, Latvia, China, Turkmenistan, Britain, Kazakhstan, South Korea, Turkey, Russia, the United States and Switzerland.

The official spoke on condition of anonymity because he was not authorized to disclose the information, which was not included in the report.

According to the report, 10 Pamir Airways pilots were paid $320,000 in salaries between March 2008 and November 2010 under the description of "pilots of cash delivery."

The report said the Afghan attorney general's office recently asked for international assistance in tracking the funds abroad but the request was limited to help from four countries ? Switzerland, France, Britain and India ? for information on funds tied to the former top two bank executives.

The report also criticizes the attorney general's office for not undertaking a substantial probe into the bank until April 2011 ? a year after the news of the bank's problems surfaced, eight months after nervous customers ran to withdraw deposits and five months after the central bank had asked the attorney general's office to start a criminal investigation.

The indictment names more than 20 bank executives, bank employees, central bank workers and others who allegedly benefited from the fraud. The charges include money laundering, misuse of authority, using counterfeit documents and opening accounts under pseudonyms. But the report claims the indictment is tainted by political influence.

"Information received during the inquiry indicates that the final decision about who to indict was made at the political level in the spring of 2011 by a high-ranking committee ? and that prosecutors from the attorney general's office were called in to amend the indictment to conform to the decisions taken," the report said.

Basir Azizi, a spokesman for the attorney general's office, said Tuesday that the Kabul Bank case was not treated as a political issue.

"We strongly reject any comments that the attorney general's office dealt with this case as a political issue," he said.

The committee also is critical of the special tribunal that Karzai created to hear the case. The report said the tribunal has had off-the-record meetings with accused individuals and potential witnesses, has conducted its own probes on the sidelines and has held meetings with shareholders, urging them to repay money ? a job tasked to the receivership.

As of Oct. 31, the receivership has recovered $135.3 million in cash as well as assets with a book value of $181.1 million, according to the committee.

The report gives the Afghan central bank credit for carrying out various examinations of the bank, saying it tried to take enforcement measures or corrective actions four times after consistently spotting regulatory violations. But the committee said that unless the central bank and other Afghan institutions move to operate independently, stand up to political interference and hold wrongdoers accountable, the Afghan government will never be able to sustain a fully functioning democracy.

The report said $861 million, or 92 percent of Kabul Bank's loan book, went to 19 individuals and companies. Among them are key bank shareholders, including Sherkhan Farnood, the former bank chairman and a world-class poker player, former chief executive officer Khalilullah Ferozi, and the brothers of Karzai and first Vice President Mohammad Qasim Fahim.

Other bank funds ? an estimated $66 million ? were spent on lavish expenses, cars, rent, bonuses, salary advances and salaries for employees that did not exist

In 2009, the central bank advised Afghan banks to refrain from making political contributions in the presidential campaign.

"This letter was not enough to dissuade Kabul Bank, which reportedly provided millions of dollars to the campaign of at least one presidential candidate ? in addition to dozens of cars, and payment of the entire media campaign including billboards and television advertisements," the report said.

___

Associated Press writer Rahim Faiez contributed to this report.

Source: http://news.yahoo.com/report-kabul-bank-sent-millions-dollars-abroad-051243748--finance.html

Phyllis Diller

Tuesday, November 27, 2012

New understanding of X chromosome inactivation

ScienceDaily (Nov. 27, 2012) ? In a paper published in the Nov. 21 issue of Cell, a team led by Mauro Calabrese, a postdoctoral fellow at the University of North Carolina in the lab of Terry Magnuson, chair of the department of genetics and member of the UNC Lineberger Comprehensive Cancer Center, broadens the understanding of how cells regulate silencing of the X chromosome in a process known as X-inactivation.

"This is a classic example of a basic research discovery. X-inactivation is a flagship model for understanding how non-coding RNAs orchestrate large-scale control of gene expression. In the simplest terms, we are trying to understand how cells regulate expression of their genes. Our findings are relevant across the board -- by understanding how normal cells function we can apply that knowledge to similar situations in the understanding and treatment of disease," said Calabrese.

Proper regulation of the X chromosome plays a crucial role in mammalian development. Females inherit a pair of X chromosomes from their parents, and the process of X-inactivation shuts down one of these two Xs.

"Males have XY. Females have two Xs. One of those Xs needs to get shut off. If it does not, it's not compatible with life. It's how we have evolved to equalize doses between males and females," said Calabrese.

While the manner in which the X chromosome is deactivated has been actively studied for 50 years, the exact mechanisms that regulate the process remain a mystery. Calabrese's research used high-throughput sequencing to determine the location and activity of chromosomes with far greater accuracy than previous research.

"Basically, this is using the sequencing technology as a high resolution microscope," said Calabrese.

Under a microscope, the inactive X chromosome (Xi) appears as a cloud-like structure, because it is covered with a non-coding RNA known as Xist. In the traditional model of X-inactivation, genes located inside the cloud are completely silenced, with 15 percent of the genes from the inactive X chromosomes escaping to become active.

"The prevailing thought was that genes that escaped X inactivation were pulled out of the core and expressed out there," said Calabrese.

The work of Calabrese's team complicates the current model of X-inactivation by finding indications of gene activity inside the Xist cloud and the presence of inactive genes outside the cloud, both of which would not have been thought possible in the prevailing model.

"It's kind of a subtle thing, but mechanistically it is a big difference," said Calabrese.

Inside the Xist cloud, sequencing discovered traces of DNase I sensitivity, a feature usually linked to transcription activity. While other markers associated with transcription were absent, the presence of DNase I sensitivity suggested that the nucleus did recognize the inactive X as usable DNA, but an unknown suppressive mechanism was preventing genes from being activated.

"We were surprised to see that. If they were totally silent, you would expect this to be not there? This suggests that transcription factors or other proteins that bind DNA are still accessing the inactive X," said Calabrese.

The other surprising findings involve the 15 percent of "escaper" genes from the inactive X. Calabrese found evidence that active genes were found both inside and outside the Xist cloud, and that silenced genes that lay alongside active genes outside of the Xist cloud remained inactive.

"If X-inactivation was a strict nuclear barrier, then pulling a gene outside the barrier would turn it on, but it has got to be more than that because when an inactivated gene that is beside an escaper is outside this domain, it is still turned off," said Calabrese.

The presence of DNase I sensitivity within the Xist cloud and the finding of inactive genes outside of the cloud suggest that a site-specific mechanism is regulating genes on the chromosome in a more subtle way than the binary "on/off" function posited by the prevailing model. The exact mechanism for this remains unknown. Although Calabrese believes that Xist still plays a role, its exact function and whether other factors influence X-inactivation remain questions for future research.

"We know that Xist is required to turn off the inactive X. We know that. We have no idea how" said Calabrese.

Beyond revising the understanding of how X-inactivation works, Calabrese said that deeper understanding of the function of Xist could reveal more about the role of other non-coding RNAs in cellular development. These RNAs could become useful targets for future therapies and drug development.

"We know that too much expression of the wrong non-coding RNAs can lead to cancer. Also, forced expression of other non-coding RNAs can prevent cancer. Generally, we do not know how these RNAs work," said Calabrese.

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Story Source:

The above story is reprinted from materials provided by University of North Carolina Health Care. The original article was written by Mary Ruth.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.


Journal Reference:

  1. J.?Mauro Calabrese, Wei Sun, Lingyun Song, Joshua?W. Mugford, Lucy Williams, Della Yee, Joshua Starmer, Piotr Mieczkowski, Gregory?E. Crawford, Terry Magnuson. Site-Specific Silencing of Regulatory Elements as a Mechanism of X Inactivation. Cell, 2012; 151 (5): 951 DOI: 10.1016/j.cell.2012.10.037

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/~3/r29Zybqw_qY/121127101534.htm

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Monday, November 26, 2012

Column: Irish smiling, but SEC eyes another title

ATHENS, Ga. (AP) ? Thanks, Notre Dame.

At least we don't have to endure another All-SEC matchup for the BCS championship.

But the conference everyone outside the South loves to hate staked out its usual place in the championship hierarchy on Super Saturday, a coast-to-coast buffet of college football's greatest rivalries.

Either Alabama or Georgia will be playing for No. 1.

Sound familiar?

The Southeastern Conference has already won an unprecedented six straight national championships, and here's betting the Crimson Tide or the Bulldogs will make it seven in a row on Jan. 7 in Miami.

Nothing against top-ranked Notre Dame, the best story in college football all season. The Fighting Irish (12-0) wrapped up their first perfect regular season since 1988 with a 22-13 victory over Southern California, leaving no doubt they have stifling defense and tremendous heart. Plus, they deserve bonus points for handing Trojans coach Lane Kiffin another loss, the perfect capper to his season-long plunge from No. 1 to irrelevance, with a few deflated footballs along the way.

All hail the Irish.

They deserve it.

For now.

"We're not done yet," Notre Dame quarterback Everett Golson said. "We've got to keep going."

Not so fast. It will be a month and a half before the champion is crowned, a ridiculously long wait in a sport where players are used to taking the field in tidy, one-week intervals. Offenses, which rely so heavily on timing and communication, tend to suffer more from these long delays, so points will be hard to come by in South Florida.

That levels the playing just a bit, since all three of the title contenders have top-shelf defenses.

Still, Alabama and Georgia both have more weapons offensively, which is why either team should have an edge on the Irish, no matter which one emerges as the winner in next Saturday's SEC championship game.

The only thing that didn't go the SEC's way on rivalry day was Notre Dame beating USC. Without the Irish gutting out another win, we very well could've had a repeat of an SEC game doubling as the title game, with Florida hoping to follow the Alabama Plan, Version 2.011.

A year ago, the Tide team didn't even win its division but got a do-over against LSU for the national title. Alabama romped in the rematch, 21-0.

From the rest of the nation, a giant yawn.

This time, the Tide cruised into the SEC championship game on a high after thumping Auburn 49-0 in the Mismatch Bowl (sorry, the Iron Bowl moniker just doesn't fit at the moment), surely bringing the Gene Chizik era to an inglorious end a mere two years after he guided the Tigers to a national championship.

Georgia will be waiting in Atlanta as the East Division winner for the second year in a row, finishing off its regular season with a 42-10 blowout of Georgia Tech. The Bulldogs are our Comeback Team of the Year, somehow finding a way to get back in the mix after taking a four-touchdown whipping at South Carolina.

"Keep up the good work," Georgia Gov. Nathan Deal told the Bulldogs after presenting a trophy for their big win over the Yellow Jackets. "We're looking for a national championship."

So was Florida, which was fourth in the BCS standings a week ago. The Gators did nothing to hurt their position, stunning Florida State with a 24-point outburst in the fourth quarter on the way to a 37-26 victory. As soon as that one ended, coach Will Muschamp and his players headed off to root for the Trojans, hoping they could take down the Irish.

It wasn't to be. Notre Dame clinched the win with a brilliant goal-line stand, stuffing the Trojans after they had first-and-goal at the 1.

Oregon was the only other team with a shot, and the Ducks took care of business with an impressive 48-24 win over Oregon State in that state's version of the Civil War. But they're all done, too. Heck, the Ducks didn't even make the Pac-12 title game; Stanford locked up the spot with a victory over UCLA.

Hard to believe, but the SEC seemed down and out just a few weeks ago. Alabama was knocked off by conference newcomer Texas A&M in Tuscaloosa, leaving three non-SEC teams ? Oregon, Kansas State and Notre Dame ? in the lead positions.

Three-fourths of the nation rejoiced.

Its long, Dixie-induced nightmare seemed over.

Of course, everything flipped again seven days later.

Oregon lost. Kansas State lost. Just like that, Alabama and Georgia were right back in the prime positions.

The Tide and the Bulldogs closed the deal Saturday against overmatched opponents, proving once again the SEC has, if nothing else, an impeccable sense of timing. Outside of Notre Dame, every team in the mix had one loss. But Oregon and Kansas State lost last, so they get shortchanged.

Ohio State might have messed things up, finishing off a 12-0 debut season for coach Urban Meyer with a victory over Michigan. But the Buckeyes are on NCAA probation because of Tattoo-gate, the championship dreams pushed off into the future by some shady ink. They look like the sort of program that can challenge the SEC's dominance, but not this year.

Maybe the new four-team playoff will stir things up a bit. Surely, it can't hurt, because the SEC clearly has this system all figured out.

"I knew the way the SEC works," Georgia defensive back Sanders Commings said, savoring his team's position in the locker room beneath Sanford Stadium. "I knew we could beat everybody else on our schedule. I was like, 'Man, we've just got to win out.'"

There will be those who say the SEC is living on its reputation, that the league isn't nearly as strong from top-to-bottom this season as it's been in other years. That's a pretty compelling argument, too. Neither Auburn nor Kentucky won an SEC game. Kentucky, Tennessee and Arkansas are looking for new coaches, and Auburn will surely be joining the list of schools with a vacancy.

But, for those at the top, there's another chance to play for a national title.

For the rest of the nation, that playoff system can't get here fast enough.

___

Paul Newberry is a national writer for The Associated Press. Write to him at pnewberry(at)ap.org and www.twitter.com/pnewberry1963

Source: http://news.yahoo.com/column-irish-smiling-sec-eyes-another-title-054659605--spt.html

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World stocks muted ahead of meeting on Greece

People stand in the rain in front of the electronic stock board of a securities firm in Tokyo, Monday, Nov. 26, 2012. Asian stock markets posted slight gains Monday after the unofficial start of the holiday shopping season in the U.S. topped expectations, offsetting concerns about Greece's financial crisis. (AP Photo/Koji Sasahara)

People stand in the rain in front of the electronic stock board of a securities firm in Tokyo, Monday, Nov. 26, 2012. Asian stock markets posted slight gains Monday after the unofficial start of the holiday shopping season in the U.S. topped expectations, offsetting concerns about Greece's financial crisis. (AP Photo/Koji Sasahara)

A woman stands in the rain in front of an electronic stock board of a securities firm in Tokyo, Monday, Nov. 26, 2012. Asian stock markets posted slight gains Monday after the unofficial start of the holiday shopping season in the U.S. topped expectations, offsetting concerns about Greece's financial crisis. (AP Photo/Koji Sasahara)

(AP) ? Asian stock markets rose modestly Monday after the unofficial start of the holiday shopping season in the U.S. topped expectations. But trading in Europe was subdued hours before finance ministers gathered yet again to discuss what to do about Greece.

The ministers of the 17 countries that use the euro are scheduled to meet in Brussels to try to reach an agreement on conditions that Greece must meet before the next installment of its emergency bailout loan can be disbursed. Athens faces bankruptcy without the cash.

In early European trading, Britain's FTSE 100 fell 0.1 percent to 5,812.32. Germany's DAX was 0.1 percent down at 7,301.64. France's CAC-40 lost 0.3 percent to 3,519.75.

Wall Street, gearing up for its first full day of trading since last Wednesday, was set to fall. Dow Jones industrial futures lost 0.2 percent at 12,937 and S&P 500 futures shed 0.3 percent to 1,401.50. U.S. stocks rose on Friday after a half-day of trading.

Stocks in Asia fared better, posting some modest gains after what appeared to be a successful start to the traditional pre-Christmas U.S. shopping season.

Americans visited stores and websites in record numbers last Friday, the day after the Thanksgiving holiday that is dubbed "Black Friday" because U.S. retailers traditionally turn a profit as millions of Americans rush out to stores in search of gifts for Christmas and other celebrations.

Surveys showed a record 247 million shoppers visited stores and websites between Thursday and Sunday, up 9.2 percent from the year before.

Japan's Nikkei 225 index rose 0.2 percent to 9,388.94 while Australia's S&P/ASX 200 added 0.3 percent to 4,424.20. Benchmarks in Singapore, Taiwan, Indonesia, India and the Philippines also rose.

But South Korea's Kospi shed 0.2 percent to 1,908.15. Hong Kong's Hang Seng was sapped of momentum by lethargic mainland Chinese markets. The index lost 0.3 percent to 21,857.77. The Shanghai Composite Index was down 0.5 percent to 2,017.46. The smaller Shenzhen Composite Index lost 1.4 percent to 789.49.

Francis Lun, managing director of Lyncean Holdings in Hong Kong, said traders were shying away from mainland stock markets due to the failure of Chinese authorities to remove companies that fail to earn profits after three years.

A regulation exists to allow for a delisting after three years, but it is not enforced, Lun said.

"If you cannot weed out the losers, the stock market will be inundated with companies not doing anything," he said. "The listed companies are out to grab money instead of earning a profit for shareholders."

Among individual stocks, Japanese vehicle makers posted solid gains. Toyota Motor Corp. rose 1.7 percent. Nissan Motor Co. added 2.3 percent. Yamaha Motor Co. gained 2.1 percent.

Australia's Sydney Airport rose 1.5 percent after announcing it has secured about $1.1 billion in new funds to repay existing debts and fund future spending.

Benchmark crude for January delivery was down 9 cents to $88.18 per barrel in electronic trading on the New York Mercantile Exchange. The contract rose 90 cents to finish at $88.28 per barrel on Friday.

In currencies, the euro fell to $1.2957 from $1.2971 late Friday in New York. The dollar fell to 82.01 yen from 82.40 yen. Earlier Monday, the dollar rose to 82.59 yen.

___

Follow Pamela Sampson on Twitter at http://twitter.com/pamelasampson

Associated Press

Source: http://hosted2.ap.org/APDEFAULT/f70471f764144b2fab526d39972d37b3/Article_2012-11-26-World%20Markets/id-6825778539634d298c49d8eef0706bb5

npr

chrisrobsonxs: pentateuch bridle: Leptospires in wildlife from ...

Everard, C. O. R.; Fraser Chanpong, G. M.; Bhagwandin, L. J.; Race, M. W.; James, A. C., 1983: Leptospires in wildlife from Trinidad and Grenada. Journal of Wildlife Diseases 19(3): 192-199

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  7. Creation and dynamics of an extracorporeal population of leptospires in wildlife foci of leptospirosis Litvin, V. Yu; Golubev, M. V., 1982: Creation and dynamics of an extracorporeal population of leptospires in wildlife foci of leptospirosis. Zhurnal Mikrobiologii Epidemiologii i Immunobiologii (3): 32-35...

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Tags: grenada, leptospires, mongooses, opossums, peridomestic

Source: http://scien.net/serum/leptospires-in-wildlife-from-trinidad-and-grenada

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ScienceDaily: Gene News

ScienceDaily: Gene Newshttp://www.sciencedaily.com/news/health_medicine/genes/ Genes and Genetics News. Read today's medical research in genetics including what can damage genes, what can protect them, and more.en-usSun, 25 Nov 2012 03:23:59 ESTSun, 25 Nov 2012 03:23:59 EST60ScienceDaily: Gene Newshttp://www.sciencedaily.com/images/logosmall.gifhttp://www.sciencedaily.com/news/health_medicine/genes/ For more science articles, visit ScienceDaily.New molecular culprit linked to breast cancer progressionhttp://www.sciencedaily.com/releases/2012/11/121124090511.htm Researchers have uncovered a protein ?partner? commonly used by breast cancer cells to unlock genes needed for spreading the disease around the body. A report on the discovery details how some tumors get the tools they need to metastasize.Sat, 24 Nov 2012 09:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121124090511.htmNew insights into virus proteome: Unknown proteins of the herpesvirus discoveredhttp://www.sciencedaily.com/releases/2012/11/121123092132.htm The genome encodes the complete information needed by an organism, including that required for protein production. Viruses, which are up to a thousand times smaller than human cells, have considerably smaller genomes. Using a type of herpesvirus as a model system scientists have shown that the genome of this virus contains much more information than previously assumed. The researchers identified several hundred novel proteins, many of which were surprisingly small.Fri, 23 Nov 2012 09:21:21 ESThttp://www.sciencedaily.com/releases/2012/11/121123092132.htmScientists describe elusive replication machinery of flu viruseshttp://www.sciencedaily.com/releases/2012/11/121122152928.htm Scientists have made a major advance in understanding how flu viruses replicate within infected cells. The researchers used cutting-edge molecular biology and electron-microscopy techniques to ?see? one of influenza?s essential protein complexes in unprecedented detail. The images generated in the study show flu virus proteins in the act of self-replication, highlighting the virus?s vulnerabilities that are sure to be of interest to drug developers.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152928.htmProtein folding: Look back on scientific advances made as result of 50-year old puzzlehttp://www.sciencedaily.com/releases/2012/11/121122152910.htm Fifty years after scientists first posed a question about protein folding, the search for answers has led to the creation of a full-fledged field of research that led to major advances in supercomputers, new materials and drug discovery, and shaped our understanding of the basic processes of life, including so-called "protein-folding diseases" such as Alzheimer's, Parkinson's and type II diabetes.Thu, 22 Nov 2012 15:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121122152910.htmStep forward in regenerating and repairing damaged nerve cellshttp://www.sciencedaily.com/releases/2012/11/121121145638.htm Researchers recently uncovered a nerve cell's internal clock, used during embryonic development. This breakthrough could lead to the development of new tools to repair and regenerate nerve cells following injuries to the central nervous system.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145638.htmArchitecture of rod sensory cilium disrupted by mutationhttp://www.sciencedaily.com/releases/2012/11/121121145621.htm Using a new technique called cryo-electron tomography, scientists have created a three-dimensional map that gives a better understanding of how the architecture of the rod sensory cilium (part of one type of photoreceptor in the eye) is changed by genetic mutation and how that affects its ability to transport proteins as part of the light-sensing process.Wed, 21 Nov 2012 14:56:56 ESThttp://www.sciencedaily.com/releases/2012/11/121121145621.htmAging: Scientists further unravel telomere biologyhttp://www.sciencedaily.com/releases/2012/11/121121130933.htm Researchers have resolved the structure of that allows a telomere-related protein, Cdc13, to form dimers in yeast. Mutations in this region of Cdc13 put the kibosh on the ability of telomerase and other proteins to maintain telomeres.Wed, 21 Nov 2012 13:09:09 ESThttp://www.sciencedaily.com/releases/2012/11/121121130933.htmDrug resistance biomarker could improve cancer treatmenthttp://www.sciencedaily.com/releases/2012/11/121121130811.htm Cancer therapies often have short-lived benefits due to the emergence of genetic mutations that cause drug resistance. A key gene that determines resistance to a range of cancer drugs has been reported in a new study. The study reveals a biomarker that can predict responses to cancer drugs and offers a strategy to treat drug-resistant tumors based on their genetic signature.Wed, 21 Nov 2012 13:08:08 ESThttp://www.sciencedaily.com/releases/2012/11/121121130811.htmGenome packaging: Key to breast cancer developementhttp://www.sciencedaily.com/releases/2012/11/121121130703.htm Two recent studies delve into the role of chromatin modifying enzymes and transcription factors in tumour cells. In one, it was found that the PARP1 enzyme activated by kinase CDK2 is necessary to induce the genes responsible for the proliferation of breast cancer cells in response to progesterone. In another, extensive work has been undertaken to identify those genes activated by the administration of progesterone in breast cancer, the sequences that can be recognized and how these genes are induced.Wed, 21 Nov 2012 13:07:07 ESThttp://www.sciencedaily.com/releases/2012/11/121121130703.htmShort DNA strands in genome may be key to understanding human cognition and diseaseshttp://www.sciencedaily.com/releases/2012/11/121121130643.htm Previously discarded, human-specific ?junk? DNA represents untapped resource in the study of diseases like Alzheimer?s and autism.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130643.htmBiomarking time: Methylome modifications offer new measure of our 'biological' agehttp://www.sciencedaily.com/releases/2012/11/121121130633.htm In a new study, researchers describe markers and a model that quantify how aging occurs at the level of genes and molecules, providing not just a more precise way to determine how old someone is, but also perhaps anticipate or treat ailments and diseases that come with the passage of time.Wed, 21 Nov 2012 13:06:06 ESThttp://www.sciencedaily.com/releases/2012/11/121121130633.htmKidney tumors have a mind of their ownhttp://www.sciencedaily.com/releases/2012/11/121121104552.htm New research has found there are several different ways that kidney tumors can achieve the same result -- namely, grow.Wed, 21 Nov 2012 10:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121121104552.htmMechanism to repair clumped proteins explainedhttp://www.sciencedaily.com/releases/2012/11/121121104416.htm Clumped proteins can be dissolved with the aid of cellular repair systems -- a process of critical importance for cell survival especially under conditions of stress. Researchers have now decrypted the fundamental mechanism for dissolving protein aggregates that involves specific molecular chaperones.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104416.htmNovel mechanism through which normal stromal cells become cancer-promoting stromal cells identifiedhttp://www.sciencedaily.com/releases/2012/11/121121104401.htm New understanding of molecular changes that convert harmless cells surrounding ovarian cancer cells into cells that promote tumor growth and metastasis provides potential new therapeutic targets for this deadly disease, according to new research.Wed, 21 Nov 2012 10:44:44 ESThttp://www.sciencedaily.com/releases/2012/11/121121104401.htmNew test for tuberculosis could improve treatment, prevent deaths in Southern Africahttp://www.sciencedaily.com/releases/2012/11/121120194932.htm A new rapid test for tuberculosis (TB) could substantially and cost-effectively reduce TB deaths and improve treatment in southern Africa -- a region where both HIV and tuberculosis are common.Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194932.htmEvolution of human intellect: Human-specific regulation of neuronal geneshttp://www.sciencedaily.com/releases/2012/11/121120194926.htm A new study has identified hundreds of small regions of the genome that appear to be uniquely regulated in human neurons. These regulatory differences distinguish us from other primates, including monkeys and apes, and as neurons are at the core of our unique cognitive abilities, these features may ultimately hold the key to our intellectual prowess (and also to our potential vulnerability to a wide range of 'human-specific' diseases from autism to Alzheimer's).Tue, 20 Nov 2012 19:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121120194926.htmRibosome regulates viral protein synthesis, revealing potential therapeutic targethttp://www.sciencedaily.com/releases/2012/11/121120132906.htm Rather than target RNA viruses directly, aiming at the host cells they invade could hold promise, but any such strategy would have to be harmless to the host. Now, a surprising discovery made in ribosomes may point the way to fighting fatal viral infections such as rabies.Tue, 20 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121120132906.htmHow does antibiotic resistance spread? Scientists find answers in the nosehttp://www.sciencedaily.com/releases/2012/11/121120121835.htm Microbiologists studying bacterial colonization in mice have discovered how the very rapid and efficient spread of antibiotic resistance works in the respiratory pathogen, Streptococcus pneumoniae (also known as the pneumococcus). The team found that resistance stems from the transfer of DNA between bacterial strains in biofilms in the nasopharynx, the area just behind the nose.Tue, 20 Nov 2012 12:18:18 ESThttp://www.sciencedaily.com/releases/2012/11/121120121835.htmScientists identify inhibitor of myelin formation in central nervous systemhttp://www.sciencedaily.com/releases/2012/11/121120100155.htm Scientists have discovered another molecule that plays an important role in regulating myelin formation in the central nervous system. Myelin promotes the conduction of nerve cell impulses by forming a sheath around their projections, the so-called axons, at specific locations -- acting like the plastic insulation around a power cord.Tue, 20 Nov 2012 10:01:01 ESThttp://www.sciencedaily.com/releases/2012/11/121120100155.htm'Obese but happy gene' challenges the common perception of link between depression and obesityhttp://www.sciencedaily.com/releases/2012/11/121120084725.htm Researchers have discovered new genetic evidence about why some people are happier than others. The scientists have uncovered evidence that the gene FTO -- the major genetic contributor to obesity -- is associated with an eight per cent reduction in the risk of depression. In other words, it's not just an obesity gene but a "happy gene" as well.Tue, 20 Nov 2012 08:47:47 ESThttp://www.sciencedaily.com/releases/2012/11/121120084725.htmTelomere lengths predict life expectancy in the wild, research showshttp://www.sciencedaily.com/releases/2012/11/121119213144.htm Researchers have found that biological age and life expectancy can be predicted by measuring an individual's DNA. They studied the length of chromosome caps -- known as telomeres -- in a 320-strong wild population of Seychelles Warblers on a small isolated island.Mon, 19 Nov 2012 21:31:31 ESThttp://www.sciencedaily.com/releases/2012/11/121119213144.htmCancer: Some cells don't know when to stophttp://www.sciencedaily.com/releases/2012/11/121119171403.htm Certain mutated cells keep trying to replicate their DNA -- with disastrous results -- even after medications rob them of the raw materials to do so, according to new research.Mon, 19 Nov 2012 17:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121119171403.htmMultiple sclerosis ?immune exchange? between brain and blood is uncoveredhttp://www.sciencedaily.com/releases/2012/11/121119163301.htm DNA sequences obtained from a handful of patients with multiple sclerosis have revealed the existence of an ?immune exchange? that allows the disease-causing cells to move in and out of the brain.Mon, 19 Nov 2012 16:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121119163301.htmFruit fly studies guide investigators to molecular mechanism frequently misregulated in human cancershttp://www.sciencedaily.com/releases/2012/11/121119132056.htm Changes in how DNA interacts with histones ?- the proteins that package DNA ?- regulate many fundamental cell activities from stem cells maturing into a specific body cell type or blood cells becoming leukemic. These interactions are governed by a biochemical tug of war between repressors and activators, which chemically modify histones signaling them to clamp down tighter on DNA or move aside and allow a gene to be expressed.Mon, 19 Nov 2012 13:20:20 ESThttp://www.sciencedaily.com/releases/2012/11/121119132056.htm3-D light switch for the brain: Device may help treat Parkinson's, epilepsy; aid understanding of consciousnesshttp://www.sciencedaily.com/releases/2012/11/121119114249.htm A new tool for neuroscientists delivers a thousand pinpricks of light to individual neurons in the brain. The new 3-D "light switch", created by biologists and engineers, could one day be used as a neural prosthesis that could treat conditions such as Parkinson's and epilepsy by using gene therapy to turn individual brain cells on and off with light.Mon, 19 Nov 2012 11:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121119114249.htmNew factor of genetic susceptibility to Alzheimer's diseasehttp://www.sciencedaily.com/releases/2012/11/121119104944.htm A large-scale international study has just discovered a gene for susceptibility to a rare disease providing evidence of the heterogeneous aetiology of Alzheimer's disease.Mon, 19 Nov 2012 10:49:49 ESThttp://www.sciencedaily.com/releases/2012/11/121119104944.htmBlood cancer gene BCL6 identified as a key factor for differentiation of nerve cells of cerebral cortexhttp://www.sciencedaily.com/releases/2012/11/121119093848.htm The cerebral cortex is the most complex structure in our brain and the seat of consciousness, emotion, motor control and language. In order to fulfill these functions, it is composed of a diverse array of nerve cells, called cortical neurons, which are affected by many neurological and neuropsychiatric diseases. Researchers have opened new perspectives on brain development and stem cell neurobiology by discovering a gene called BCL6 as a key factor in the generation of cortical neurons during embryonic brain development.Mon, 19 Nov 2012 09:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121119093848.htmMinority report: Insight into subtle genomic differences among our own cellshttp://www.sciencedaily.com/releases/2012/11/121118141530.htm Scientists have demonstrated that induced pluripotent stem cells -- the embryonic-stem-cell look-alikes whose discovery a few years ago won this year's Nobel Prize in medicine -- are not as genetically unstable as was thought.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141530.htmSkin cells reveal DNA's genetic mosaichttp://www.sciencedaily.com/releases/2012/11/121118141524.htm The prevailing wisdom has been that every cell in the body contains identical DNA. However, a new study of stem cells derived from the skin has found that genetic variations are widespread in the body's tissues, a finding with profound implications for genetic screening.Sun, 18 Nov 2012 14:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121118141524.htmLikely basis of birth defect causing premature skull closure in infants identifiedhttp://www.sciencedaily.com/releases/2012/11/121118141432.htm Geneticists, pediatricians, surgeons and epidemiologists have identified two areas of the human genome associated with the most common form of non-syndromic craniosynostosis premature closure of the bony plates of the skull.Sun, 18 Nov 2012 14:14:14 ESThttp://www.sciencedaily.com/releases/2012/11/121118141432.htmDNA packaging discovery reveals principles by which CRC mutations may cause cancerhttp://www.sciencedaily.com/releases/2012/11/121117184658.htm A new discovery concerning a fundamental understanding about how DNA works will produce a "180-degree change in focus" for researchers who study how gene packaging regulates gene activity, including genes that cause cancer and other diseases.Sat, 17 Nov 2012 18:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121117184658.htmHepatitis C treatment's side effects can now be studied in the labhttp://www.sciencedaily.com/releases/2012/11/121116161059.htm Adverse side effects of certain hepatitis C medications can now be replicated in the lab, thanks to a research team. The new method aids understanding of recent failures of hepatitis C antiviral drugs in some patients, and could help to identify medications that eliminate adverse effects. The findings may aid the development of safer and more effective treatments for hepatitis C and other pathogens such as SARS and West Nile virus.Fri, 16 Nov 2012 16:10:10 ESThttp://www.sciencedaily.com/releases/2012/11/121116161059.htmReconsidering cancer's bad guyhttp://www.sciencedaily.com/releases/2012/11/121116124644.htm Researchers have found that a protein, known for causing cancer cells to spread around the body, is also one of the molecules that trigger repair processes in the brain.Fri, 16 Nov 2012 12:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121116124644.htmGene distinguishes early birds from night owls and helps predict time of deathhttp://www.sciencedaily.com/releases/2012/11/121116124551.htm New research shows that a gene is responsible for a person's tendency to be an early riser or night owl -- and helps determine the time of day a person is most likely to die.Fri, 16 Nov 2012 12:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121116124551.htmClues to cause of kids' brain tumorshttp://www.sciencedaily.com/releases/2012/11/121116091226.htm Insights from a genetic condition that causes brain cancer are helping scientists better understand the most common type of brain tumor in children.Fri, 16 Nov 2012 09:12:12 ESThttp://www.sciencedaily.com/releases/2012/11/121116091226.htmArthritis study reveals why gender bias is all in the geneshttp://www.sciencedaily.com/releases/2012/11/121115210541.htm Researchers have pieced together new genetic clues to the arthritis puzzle in a study that brings potential treatments closer to reality and could also provide insights into why more women than men succumb to the disabling condition.Thu, 15 Nov 2012 21:05:05 ESThttp://www.sciencedaily.com/releases/2012/11/121115210541.htmClass of RNA molecules protects germ cells from damagehttp://www.sciencedaily.com/releases/2012/11/121115172255.htm Passing one's genes on to the next generation is a mark of evolutionary success. So it makes sense that the body would work to ensure that the genes the next generation inherits are exact replicas of the originals. Biologists have now identified one way the body does exactly that.Thu, 15 Nov 2012 17:22:22 ESThttp://www.sciencedaily.com/releases/2012/11/121115172255.htmQuick test speeds search for Alzheimer's drugs: Compound restores motor function and longevity to fruit flieshttp://www.sciencedaily.com/releases/2012/11/121115152655.htm Researchers report that an efficient, high-volume technique for testing potential drug treatments for Alzheimer's disease uncovered an organic compound that restored motor function and longevity to fruit flies with the disease.Thu, 15 Nov 2012 15:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115152655.htmProtein-making machinery can switch gears with a small structural change process; Implications for immunity and cancer therapyhttp://www.sciencedaily.com/releases/2012/11/121115133414.htm For the past several years, research has focused on the intricate actions of an ancient family of catalytic enzymes that play a key role in translation, the process of producing proteins. In a new study, scientists have shown that this enzyme can actually also work in another fundamental process in humans.Thu, 15 Nov 2012 13:34:34 ESThttp://www.sciencedaily.com/releases/2012/11/121115133414.htmPlant derivative, tanshinones, protects against sepsis, study suggestshttp://www.sciencedaily.com/releases/2012/11/121115133312.htm Researchers have discovered that tanshinones, which come from the plant Danshen and are highly valued in Chinese traditional medicine, protect against the life-threatening condition sepsis.Thu, 15 Nov 2012 13:33:33 ESThttp://www.sciencedaily.com/releases/2012/11/121115133312.htmStructure of enzyme topoisomerase II alpha unravelled providing basis for more accurate design of chemotherapeutic drugshttp://www.sciencedaily.com/releases/2012/11/121115132903.htm Medical researchers have for the first time described the structure of the active site core of topoisomerase II alpha, an important target for anti-cancer drugs. The type II topoisomerases are important enzymes that are involved in maintaining the structure of DNA and chromosome segregation during both replication and transcription of DNA. One of these enzymes, topoisomerase II alpha, is involved in the replication of DNA and cell proliferation, and is highly expressed in rapidly dividing cancer cells.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132903.htmNewly discovered enzyme important in the spreading of cancerhttp://www.sciencedaily.com/releases/2012/11/121115132901.htm Enzyme hunters at UiO have discovered the function of an enzyme that is important in the spreading of cancer. Cancer researchers now hope to inhibit the enzyme.Thu, 15 Nov 2012 13:29:29 ESThttp://www.sciencedaily.com/releases/2012/11/121115132901.htmGenetics point to serious pregnancy complication, pre-eclampsiahttp://www.sciencedaily.com/releases/2012/11/121115132613.htm New research has revealed a genetic link in pregnant moms - and their male partners - to pre-eclampsia, a life-threatening complication during pregnancy.Thu, 15 Nov 2012 13:26:26 ESThttp://www.sciencedaily.com/releases/2012/11/121115132613.htmMolecular mechanisms underlying stem cell reprogramming decodedhttp://www.sciencedaily.com/releases/2012/11/121115132344.htm Thanks to some careful detective work, scientist better understand just how iPS cells form ? and why the Yamanaka process is inefficient, an important step to work out for regenerative medicine. The findings uncover cellular impediments to iPS cell development that, if overcome, could dramatically improve the efficiency and speed of iPS cell generation.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132344.htmSurprising genetic link between kidney defects and neurodevelopmental disorders in kidshttp://www.sciencedaily.com/releases/2012/11/121115132342.htm About 10 percent of kids born with kidney defects have large alterations in their genomes known to be linked with neurodevelopmental delay and mental illness, a new study has shown.Thu, 15 Nov 2012 13:23:23 ESThttp://www.sciencedaily.com/releases/2012/11/121115132342.htmEven moderate drinking in pregnancy can affect a child's IQhttp://www.sciencedaily.com/releases/2012/11/121114172833.htm Relatively small levels of exposure to alcohol while in the womb can influence a child's IQ, according to a new study using data from over 4,000 mothers and their children.Wed, 14 Nov 2012 17:28:28 ESThttp://www.sciencedaily.com/releases/2012/11/121114172833.htmGene nearly triples risk of Alzheimer's, international research team findshttp://www.sciencedaily.com/releases/2012/11/121114171710.htm A gene so powerful it nearly triples the risk of Alzheimer's disease has been discovered by an international team of researchers. It is the most potent genetic risk factor for Alzheimer's identified in the past 20 years.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171710.htmDiscovery could lead to faster diagnosis for some chronic fatigue syndrome caseshttp://www.sciencedaily.com/releases/2012/11/121114171708.htm For the first time, researchers have landed on a potential diagnostic method to identify at least a subset of patients with chronic fatigue syndrome - testing for antibodies linked to latent Epstein-Barr virus reactivation.Wed, 14 Nov 2012 17:17:17 ESThttp://www.sciencedaily.com/releases/2012/11/121114171708.htmResearch breakthrough could halt melanoma metastasis, study suggestshttp://www.sciencedaily.com/releases/2012/11/121114153227.htm In laboratory experiments, scientists have eliminated metastasis, the spread of cancer from the original tumor to other parts of the body, in melanoma by inhibiting a protein known as melanoma differentiation associated gene-9 (mda-9)/syntenin.Wed, 14 Nov 2012 15:32:32 ESThttp://www.sciencedaily.com/releases/2012/11/121114153227.htmPig genomes provide massive amount of genomic data for human healthhttp://www.sciencedaily.com/releases/2012/11/121114134512.htm Researchers provide a whole-genome sequence and analysis of number of pig breeds, including a miniature pig that serves a model for human medical studies and therapeutic drug testing.Wed, 14 Nov 2012 13:45:45 ESThttp://www.sciencedaily.com/releases/2012/11/121114134512.htmRare parasitic fungi could have anti-flammatory benefitshttp://www.sciencedaily.com/releases/2012/11/121114134054.htm Caterpillar fungi are rare parasites found on hibernating caterpillars in the mountains of Tibet. For centuries they have been highly prized as a traditional Chinese medicine - just a small amount can fetch hundreds of dollars.Wed, 14 Nov 2012 13:40:40 ESThttp://www.sciencedaily.com/releases/2012/11/121114134054.htmCancer therapy: Nanokey opens tumors to attackhttp://www.sciencedaily.com/releases/2012/11/121114113803.htm There are plenty of effective anticancer agents around. The problem is that, very often, they cannot gain access to all the cells in solid tumors. A new gene delivery vehicle may provide a way of making tracks to the heart of the target.Wed, 14 Nov 2012 11:38:38 ESThttp://www.sciencedaily.com/releases/2012/11/121114113803.htmHigh sperm DNA damage a leading cause of 'unexplained infertility', research findshttp://www.sciencedaily.com/releases/2012/11/121114113235.htm New research has uncovered the cause of infertility for 80 per cent of couples previously diagnosed with 'unexplained infertility': high sperm DNA damage.Wed, 14 Nov 2012 11:32:32 ESThttp://www.sciencedaily.com/releases/2012/11/121114113235.htmA risk gene for cannabis psychosishttp://www.sciencedaily.com/releases/2012/11/121114083928.htm The ability of cannabis to produce psychosis has long been an important public health concern. This concern is growing in importance as there is emerging data that cannabis exposure during adolescence may increase the risk of developing schizophrenia, a serious psychotic disorder. Further, with the advent of medical marijuana, a new group of people with uncertain psychosis risk may be exposed to cannabis.Wed, 14 Nov 2012 08:39:39 ESThttp://www.sciencedaily.com/releases/2012/11/121114083928.htmBacterial DNA sequence used to map an infection outbreakhttp://www.sciencedaily.com/releases/2012/11/121113214635.htm For the first time, researchers have used DNA sequencing to help bring an infectious disease outbreak in a hospital to a close. Researchers used advanced DNA sequencing technologies to confirm the presence of an ongoing outbreak of methicillin-resistant Staphylococcus aureus (MRSA) in a Special Care Baby Unit in real time. This assisted in stopping the outbreak earlier, saving possible harm to patients. This approach is much more accurate than current methods used to detect hospital outbreaks.Tue, 13 Nov 2012 21:46:46 ESThttp://www.sciencedaily.com/releases/2012/11/121113214635.htmGenetic variation may modify associations between low vitamin D levels and adverse health outcomeshttp://www.sciencedaily.com/releases/2012/11/121113161506.htm Findings from a study suggest that certain variations in vitamin D metabolism genes may modify the association of low serum 25-hydroxyvitamin D concentrations with health outcomes such as hip fracture, heart attack, cancer, and death.Tue, 13 Nov 2012 16:15:15 ESThttp://www.sciencedaily.com/releases/2012/11/121113161506.htmNew type of bacterial protection found within cells: Novel immune system response to infections discoveredhttp://www.sciencedaily.com/releases/2012/11/121113143656.htm Biologists have discovered that fats within cells store a class of proteins with potent antibacterial activity, revealing a previously unknown type of immune system response that targets and kills bacterial infections.Tue, 13 Nov 2012 14:36:36 ESThttp://www.sciencedaily.com/releases/2012/11/121113143656.htmGlutamate neurotransmission system may be involved with depression riskhttp://www.sciencedaily.com/releases/2012/11/121113134807.htm Researchers using a new approach to identifying genes associated with depression have found that variants in a group of genes involved in transmission of signals by the neurotransmitter glutamate appear to increase the risk of depression.Tue, 13 Nov 2012 13:48:48 ESThttp://www.sciencedaily.com/releases/2012/11/121113134807.htmTargeting downstream proteins in cancer-causing pathway shows promise in cell, animal modelhttp://www.sciencedaily.com/releases/2012/11/121113134230.htm The cancer-causing form of the gene Myc alters the metabolism of mitochondria, the cell?s powerhouse, making it dependent on the amino acid glutamine for survival. Depriving cells of glutamine selectively induces programmed cell death in cells overexpressing mutant Myc. Using Myc-active neuroblastoma cells, a team three priotein executors of the glutamine-starved cell, representing a downstream target at which to aim drugs. Roughly 25 percent of all neuroblastoma cases are associated with Myc-active cells.Tue, 13 Nov 2012 13:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121113134230.htmEven low-level radioactivity is damaging, scientists concludehttp://www.sciencedaily.com/releases/2012/11/121113134224.htm Even the very lowest levels of radiation are harmful to life, scientists have concluded, reporting the results of a wide-ranging analysis of 46 peer-reviewed studies published over the past 40 years. Variation in low-level, natural background radiation was found to have small, but highly statistically significant, negative effects on DNA as well as several measures of health.Tue, 13 Nov 2012 13:42:42 ESThttp://www.sciencedaily.com/releases/2012/11/121113134224.htm

Source: http://www.sciencedaily.com/rss/health_medicine/genes.xml

justin beiber

President Mahama says only NDC has a cogent programme on ...

President John Dramani Mahama says it is only the National Democratic Congress (NDC) that has a cogent programme on education which links up the various levels on the educational ladder.

He said the NDC planned to expand access at the basic level, secondary and tertiary levels.

President Mahama was addressing students of the Koforidua Polytechnic who cheered him on as he addressed them.

President Mahama, who is the Presidential candidate of the NDC for the 2012 election, had earlier addressed rallies at Atimpoku, Odumase Krobo, Adukrom, Adawso and Begoro.

People lined up the streets in all the areas to catch a glimpse of the President and cheer him on. Some among the crowd danced ?Azonto? to the NDC?s songs.

The President said the next NDC government would continue with efforts to make basic education truly free and universal and take steps to make SHS progressively accessible and free.

He said the next NDC government would build 200 community senior high schools (SHS) to accommodate more students from the Junior High Schools (JHS).

Besides, he said, the government would put up 10 colleges of education, build more vocational and technical schools, build more nursing and midwifery training colleges and establish one public university in the Eastern Region.

The President said the expansion of facilities at the various levels of the educational ladder would allow students to progress from one stage of the educational ladder to the other without any hitches.

On polytechnic education, he said the next NDC government would improve faculties and expand infrastructure at the various polytechnics to make it possible for the polytechnics to award their own degrees.

Under that arrangement, he said polytechnic students would be able to acquire their own degrees without affiliation to any universities.

President Mahama said the NDC could be trusted to deliver on its promises because it had a track record of delivery.

For instance, he said, it was the first NDC government that established the University of Education, Winneba, the University of Mines and Technology, Tarkwa, and the University for Development Studies (UDS) in the north.

Besides, he said, the previous NDC government established 119 SHS, seven polytechnics and several vocational and technical schools.

The President said the current NDC government has established the University of Health and Allied Sciences in the Volta Region and the University of Energy and Natural Resources in the Brong Ahafo Region, indicating that both universities had just matriculated their first batches of students.

He, therefore, asked Ghanaians to discount the NPP?s proposed free SHS since it was a m ere political gimmick to get Ghanaians into voting for the party.

President Mahama contended that the NDC government?s performance in the areas of education, healthcare, economy and infrastructure development in the last four years had been remarkable.

He said the NDC government used the period to lay the foundation.

Therefore, he said, it was crucial for Ghanaians to renew the mandate of the NDC to build on what it had started.

Source:?Daily Graphic

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  4. Group lauds President Mahama?s performance at IEA Evening Encounter ?Accra, Oct.23, GNA ? The Young Democrats (TYDE), a network...
  5. President Mahama: NPP?s promise to build 350 SHS is deceitful President John Mahama has described the NPP?s promise of building...
  6. Polytechnics Will Soon Become Technical Univarsities ? Mahama President?John Dramani Mahama?says his government, when retained in power in...
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Source: http://www.spyghana.com/politics/president-mahama-says-only-ndc-has-a-cogent-programme-on-education/

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Sunday, November 25, 2012

State Department wonders why Egyptians can?t get along (Powerlineblog)

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rigidify gazpacho: Hemorrhoids Relief Natural ? Health & Fitness ...

Linda asks?If two independent experiments failed to replicate the Abiogenesis Theory does it falsify the Theory?In natural science, abiogenesis or biopoesis is the study of how biological life arises from inorganic matter. (Not ignorance matters, That?s an atheist fundie term)

The two independently conducted experiments one by an agnostic and the other by an atheist team both failed miserably.

The agnostic bashed two rocks together in pond scum and failed to make life or dinosaurs.

While

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